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Convert height between centimetres and feet & inches, find your height percentile by age and gender, and predict your child’s adult height using the clinically validated Mid-Parental Height method.
Enter your height in either unit below — the other updates automatically.
All equivalent measurements at a glance
Height is one of the most heritable human traits — approximately 80% of adult height variation is genetic. The remaining 20% is shaped by nutrition, sleep, hormones, and early-life health. Understanding where your height falls relative to population norms is useful context for health and growth monitoring.
Your height percentile shows what percentage of people of the same age and gender are shorter than you. The 50th percentile is the statistical average. The 25th–75th range is considered typical.
Most people reach their adult height by 16–18 (females) or 18–21 (males), when growth plates (epiphyses) close. After this, height remains stable until age-related compression in older adults.
About 80% of height is determined by multiple genes inherited from both parents. The Mid-Parental Height formula captures this, explaining why tall parents tend to have tall children.
In growing children, adequate protein, calcium, vitamin D, zinc, and consistent quality sleep are essential for achieving genetic height potential. Chronic illness or malnutrition can limit growth.
International height averages (WHO/CDC reference data) for adult populations. Note that averages vary significantly by country and ethnicity.
| Region | Avg Height |
|---|---|
| Netherlands | 182.9 cm / 6'0" |
| Australia | 175.6 cm / 5'9" |
| USA | 175.4 cm / 5'9" |
| India | 165.0 cm / 5'5" |
| Global Average | 171.0 cm / 5'7" |
| Region | Avg Height |
|---|---|
| Netherlands | 170.4 cm / 5'7" |
| Australia | 162.0 cm / 5'4" |
| USA | 161.3 cm / 5'3" |
| India | 152.6 cm / 5'0" |
| Global Average | 159.5 cm / 5'3" |
Adult height is the product of genetics, environment, and timing. While you cannot change your genetics, the environmental factors below are modifiable — especially during childhood and adolescence.
Over 700 genetic variants influence height. The Mid-Parental Height formula provides a practical estimate but cannot capture all genetic complexity. Tall or short stature can come from either parent’s family line.
Adequate protein, calcium, phosphorus, vitamin D, and zinc are critical during growth phases. Chronic undernutrition — especially in the first 1,000 days of life — can permanently limit height potential.
Growth hormone (GH) is released primarily during deep (slow-wave) sleep. Children and adolescents need 9–11 hours of quality sleep per night to support optimal GH secretion and bone growth.
Weight-bearing exercise stimulates bone remodelling and GH release. Moderate exercise benefits growth; extreme endurance training in young athletes (especially females) can suppress hormones and delay growth.
Growth hormone, IGF-1, thyroid hormone, and sex hormones (oestrogen, testosterone) all regulate bone growth. Deficiencies — detectable by a paediatrician — can be treated during the growth window.
Chronic illness, repeated infections, stress, and socioeconomic factors can limit growth. Children raised in lower-income environments with limited food security consistently show reduced height-for-age versus genetic potential.
Height gain is not linear — it occurs in distinct phases, each with its own rate, drivers, and critical nutritional needs.
The fastest post-birth growth phase. Almost entirely nutrition-driven; breastfeeding and adequate micronutrients are critical.
Steady, predictable growth driven by GH. Nutrition, sleep, and overall health determine whether children track their genetic height potential.
Peak height velocity (growth spurt). Girls typically start 2 years earlier; boys reach a taller final height due to a longer pre-pubertal and pubertal growth period.
Growth plates close (epiphyseal fusion). Minimal height gain possible from residual cartilage. Bone density continues to increase until ~30.
The Mid-Parental Height (MPH) formula is the standard method used by paediatricians to estimate a child’s genetic height potential from parental heights. It is endorsed by the American Academy of Pediatrics and used in growth monitoring worldwide.
+13 cm accounts for the average male-female height difference
−13 cm accounts for the average male-female height difference
Quick-reference table converting common heights from centimetres to feet & inches. Useful when communicating with international contacts or medical professionals who use different units.
In most cases, no. Once the growth plates (epiphyseal plates) close — typically by age 18 in females and 21 in males — bone length cannot increase. Small variations in perceived height can occur throughout the day (up to 1–2 cm) due to spinal disc compression from gravity. After age 40, gradual height loss of 0.5–1 cm per decade is common due to spinal disc thinning and postural changes.
The Mid-Parental Height (MPH) formula predicts a child’s adult height within ±10 cm for approximately 95% of children. However, it cannot account for all the genetic factors, de novo mutations, or environmental influences that affect actual height. A paediatrician tracking a child’s growth trajectory on a standardised growth chart provides a far more personalised prediction over time.
For adults, height itself is rarely a primary health concern. However, being below the 3rd or above the 97th percentile for height — especially in children — may warrant investigation for underlying hormonal, genetic, or nutritional conditions. Extreme short stature can be associated with conditions such as growth hormone deficiency, hypothyroidism, or Turner syndrome. Unusual tall stature may indicate Marfan syndrome or pituitary gigantism. In adults, height differences are mostly cosmetic and social in nature.
BMI (Body Mass Index) uses height as a core input: BMI = weight (kg) ÷ height (m)². Taller individuals have a larger denominator, so the same weight gives a lower BMI for a taller person. This means tall individuals are less likely to be classified as overweight by BMI alone, while shorter individuals may be classified as overweight at lower absolute weights. This is one limitation of BMI as a universal health screening tool.
Not necessarily. While tall parents have tall children more often than average (the MPH formula captures this), there are important nuances. First, children regress toward the population mean — meaning very tall parents tend to have children who are slightly shorter than them on average. Second, the MPH range of ±10 cm is large. Third, nutrition, health, and sleep during childhood can significantly affect whether a child meets their genetic height potential.
The formula is: cm = (feet × 12 + inches) × 2.54. For example, 5 feet 8 inches = (5 × 12 + 8) × 2.54 = 68 × 2.54 = 172.7 cm. To go the other way: divide total cm by 2.54 to get total inches, then divide by 12 to get feet with the remainder as inches. Our Height Converter above does this automatically.
Consult a paediatrician if your child: (1) falls below the 3rd percentile for height-for-age; (2) consistently grows less than 4–5 cm per year during childhood; (3) has not started puberty by age 14 (boys) or 13 (girls); (4) has a growth rate that has slowed dramatically over 6–12 months; or (5) their height trajectory has deviated significantly from the MPH prediction. Early intervention for hormonal or nutritional causes of short stature can be effective within the growth window.
Whether you’re converting units, understanding your height percentile, or planning ahead for your child’s growth, our Height Calculator gives you the tools to make sense of height data in seconds.
For children still in their growth window, the most impactful investments are consistent nutrition, adequate sleep, regular physical activity, and addressing any underlying health conditions early. For adults, height is largely fixed — but pairing it with weight, waist circumference, and body composition gives a far richer picture of health than any single measurement.
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